Daryadel, A.* et al. The calcium-sensing receptor has only a parathyroid hormone-dependent role in the acute response of renal phosphatetransporters to phosphate intake. Am. J. Physiol.-Renal Physiol. 326, F792-F801 (2024) Bohnert, B.N. et al. Experimental nephrotic syndrome leads to proteolytic activation of the epithelial sodium channel (ENaC) in the mouse kidney. Am. J. Physiol.-Renal Physiol. 321, F480-F493 (2021) Fischereder, M.* et al. Sodium storage in human tissues is mediated by glycosaminoglycan expression. Am. J. Physiol.-Renal Physiol. 313, F319-F325 (2017) Mulay, S.R.* et al. Oxalate-induced chronic kidney disease with its uremic and cardiovascular complications in C57BL/6 mice. Am. J. Physiol.-Renal Physiol. 310, F785-F795 (2016) Kemter, E.* et al. Mutation of the Na⁺-K⁺-2Cl‾ cotransporter NKCC2 in mice is associated with severe polyuria and a urea-selective concentrating defect without hyperreninemia. Am. J. Physiol.-Renal Physiol. 298, F1405-F1415 (2010) Kemter, E.* et al. Novel missense mutation of uromodulin in mice causes renal dysfunction with alterations in urea handling, energy, and bone metabolism. Am. J. Physiol.-Renal Physiol. 297, F1391-F1398 (2009) Aigner, B.* et al. Screening for increased plasma urea levels in a large-scale ENU mouse mutagenesis project reveals kidney disease models. Am. J. Physiol.-Renal Physiol. 292, 1560-1567 (2007)