OpenSSL SSL_connect: Connection reset by peer in connection to v2.sherpa.ac.uk:443 PuSH - Publikationsserver des Helmholtz Zentrums München: The relationship between breast milk leptin and adiponectin with child body composition from 3 to 5 years: A follow-up study.

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Meyer, D.M.* ; Brei, C.* ; Stecher, L.* ; Much, D. ; Brunner, S.* ; Hauner, H.*

The relationship between breast milk leptin and adiponectin with child body composition from 3 to 5 years: A follow-up study.

Pediatr. Obes. 12, 1, 125-129 (2017)
DOI PMC
Open Access Green möglich sobald Postprint bei der ZB eingereicht worden ist.
BackgroundResearch indicates that breast milk contains bioactive components that influence metabolism in infancy and may play a role in the prevention of obesity in early childhood. In our initial study, 147 breastfeeding mother/child pairs were followed from birth to 2 years of age to examine the relationship between breast milk leptin and total adiponectin (collected at 6 weeks and 4 months postpartum) and infant body composition. Higher breast milk total adiponectin was related to greater fat mass and weight gain in children at 1 and 2 years of age, whereas leptin showed no association. Objectives/MethodsIn this follow-up, we examined the relationship between both adipokines and children's body weight, body mass index percentiles, sum of four skin-folds, percentage of body fat, fat mass and lean body mass at 3, 4 and 5 years of age. ResultsBreast milk adipokines were largely unrelated to child anthropometric measures. ConclusionOur results do not provide significant evidence that breast milk adipokines can predict adiposity in preschool children.
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Publikationstyp Artikel: Journalartikel
Dokumenttyp Wissenschaftlicher Artikel
Korrespondenzautor
Schlagwörter Adipokines ; Adiponectin ; Breast Milk ; Childhood Obesity; Randomized Controlled-trial; Chain Pufa Ratio; 5 Y; Pregnancy; Lactation; Adipokines; Overweight; Insulin; Growth; Design
ISSN (print) / ISBN 2047-6302
e-ISSN 2047-6310
Zeitschrift Pediatric obesity
Quellenangaben Band: 12, Heft: , Seiten: 125-129, Artikelnummer: , Supplement: 1
Verlag Wiley
Verlagsort Oxford
Nichtpatentliteratur Publikationen
Begutachtungsstatus Peer reviewed
Institut(e) Institute of Diabetes Research (IDF)