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1.
Halon-Gołąbek, M.* et al.: Amyotrophic lateral sclerosis associated disturbance of iron metabolism is blunted by swim training-role of AKT signaling pathway. Biochim. Biophys. Acta-Mol. Basis Dis. 1870:167014 (2024)
2.
Jacobs, H.T.* et al.: AOX delays the onset of the lethal phenotype in a mouse model of Uqcrh (complex III) disease. Biochim. Biophys. Acta-Mol. Basis Dis. 1869:166760 (2023)
3.
Bauer, A. et al.: Proteomics reveals antiviral host response and NETosis during acute COVID-19 in high-risk patients. Biochim. Biophys. Acta-Mol. Basis Dis. 1869:166592 (2022)
4.
Lucienne, M.* et al.: In-depth phenotyping reveals common and novel disease symptoms in a hemizygous knock-in mouse model (Mut-ko/ki) of mut-type methylmalonic aciduria. Biochim. Biophys. Acta-Mol. Basis Dis. 1866:165622 (2020)
5.
Moellmann, J.* et al.: Empagliflozin improves left ventricular diastolic function of db/db mice. Biochim. Biophys. Acta-Mol. Basis Dis. 1866:165807 (2020)
6.
Jensen, L.R.* et al.: A mouse model for intellectual disability caused by mutations in the X-linked 2 '-O-methyltransferase Ftsj1 gene. Biochim. Biophys. Acta-Mol. Basis Dis. 1865, 2083-2093 (2018)
7.
Gimpfl, M.* et al.: Modification of the fatty acid composition of an obesogenic diet improves the maternal and placental metabolic environment in obese pregnant mice. Biochim. Biophys. Acta-Mol. Basis Dis. 1863, 1605-1614 (2017)
8.
Dahlhoff, M.* et al.: Peri-conceptional obesogenic exposure induces sex-specific programming of disease susceptibilities in adult mouse offspring. Biochim. Biophys. Acta-Mol. Basis Dis. 1842, 304–317 (2014)
9.
Häbig, K.* et al.: LRRK2 guides the actin cytoskeleton at growth cones together with ARHGEF7 and Tropomyosin 4. Biochim. Biophys. Acta-Mol. Basis Dis. 1832, 2352-2367 (2013)
10.
Netzer, C.* ; Bohlander, S.K. ; Hinzke, M.* ; Chen, Y. & Kohlhase, J.*: Defining the heterochromatin localization and repression domains of SALL1. Biochim. Biophys. Acta-Mol. Basis Dis. 1762, 386-391 (2006)
11.
Kerler, F. ; Hübner, C. ; Erfle, V. & Buff, K.: Exposure of HIV-infected cells to phospholipid leads to membrane alterations and selective growth retardation. Biochim. Biophys. Acta-Mol. Basis Dis. 1139, 57-64 (1992)
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