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1.
Seizer, P.* et al.: Cyclophilin A is a ligand for RAGE in thrombo-inflammation. Cardiovasc. Res. 120, 385-402 (2024)
2.
Wang, Y.* et al.: Myeloid cannabinoid CB1 receptor deletion confers atheroprotection in male mice by reducing macrophage proliferation in a sex-dependent manner. Cardiovasc. Res.:cvae125 (2024)
3.
Nogal, A.* et al.: Predictive metabolites for incident myocardial infarction: A two-step meta-analysis of individual patient data from six cohorts comprising 7,897 individuals from the the COnsortium of METabolomic Studies. Cardiovasc. Res. 119, 2743-2754 (2023)
4.
Egea, V.* et al.: Properties and fate of human mesenchymal stem cells upon miRNA let-7f-promoted recruitment to atherosclerotic plaques. Cardiovasc. Res., DOI: 10.1093/cvr/cvac022 (2022)
5.
Immler, R.* et al.: The voltage-gated potassium channel KV1.3 regulates neutrophil recruitment during inflammation. Cardiovasc. Res. 118, 1289–1302 (2022)
6.
Zeng, L.* et al.: Cis-epistasis at the LPA locus and risk of cardiovascular diseases. Cardiovasc. Res. 118, 1088–1102 (2022)
7.
Portero, V.* et al.: Chronically elevated branched chain amino acid levels are pro-arrhythmic. Cardiovasc. Res. 118:1742–1757 (2021)
8.
Heitmeier, T.* et al.: Altered proteasome function in right ventricular hypertrophy. Cardiovasc. Res. 116, 406-415 (2020)
9.
Pinkert, S.* et al.: Development of a new mouse model for coxsackievirus-inducedmyocarditis by attenuating coxsackievirus B3 virulence in the pancreas. Cardiovasc. Res. 116, 1756-1766 (2020)
10.
Ketelhuth, D.F.J.* et al.: Immunometabolism and atherosclerosis: perspectives and clinical significance- A position paper from the working group on atherosclerosis and vascular biology of the European Society of Cardiology. Cardiovasc. Res. 115, 1385-1392 (2019)
11.
Ventura-Clapier, R.* et al.: Sex in basic research - Concepts in the cardiovascular field. Cardiovasc. Res. 113, 711-724 (2017)
12.
Legchenko, E.* ; Küffner, R. & Hansmann, G.*: Differential transcriptome and microRNA expression signatures in the healthy heart (RV vs. LV) and the failing, pressure-overloaded right ventricle (SuHx model). Cardiovasc. Res. 111, 1, S43-S43 (2016)
13.
Behr, E.R.* et al.: Role of common and rare variants in SCN10A: Results from the Brugada syndrome QRS locus gene discovery collaborative study. Cardiovasc. Res. 106, 520-529 (2015)
14.
Laschkolnig, A.* et al.: Lipoprotein (a) concentrations, apolipoprotein (a) phenotypes, and peripheral arterial disease in three independent cohorts. Cardiovasc. Res. 103, 28-36 (2014)
15.
David, R.* et al.: Induction of MesP1 by Brachyury(T) generates the common multipotent cardiovascular stem cell. Cardiovasc. Res. 92, 115-122 (2011)
16.
Sinner, M.F.* ; Ellinor, P.T.* ; Meitinger, T. ; Benjamin, E.J.* & Kääb, S.*: Genome-wide association studies of atrial fibrillation: Past, present, and future. Cardiovasc. Res. 89, 701-709 (2011)
17.
Luchner, A.* et al.: Gender-specific differences of cardiac remodeling in subjects with left ventricular dysfunction : A population-based study. Cardiovasc. Res. 53, 720-727 (2002)
18.
Hengstenberg, C.* et al.: Association between a polymorphism in the G protein ß3 subunit gene (GNB3) with arterial hypertension but not with myocardial infarction. Cardiovasc. Res. 49, 820-827 (2001)
19.
Holmer, S.R.* et al.: Lipoprotein lipase gene polymorphism, cholesterol subfractions and myocardial infarction in large samples of the general population. Cardiovasc. Res. 47, 806-812 (2000)
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