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1.
Dean-Ben, X.L. et al.: Deep tissue volumetric optoacoustic tracking of individual circulating tumor cells in an in intracardially perfused mouse model. Neoplasia 22, 441-446 (2020)
2.
Lafci, B.* ; Mercep, E. ; Herraiz, J.L.* ; Deán-Ben, X.L.* & Razansky, D.: Noninvasive multiparametric charac-terization of mammary tumors with transmission-reflection optoacoustic ultrasound. Neoplasia 22, 770-777 (2020)
3.
Ma, X. et al.: Integrin-targeted hybrid fluorescence molecular tomography/X-ray computed tomography for imaging tumor progression and early response in non-small cell lung cancer. Neoplasia 19, 8-16 (2017)
4.
Chekkoury, A. et al.: High-resolution multispectral optoacoustic tomography of the vascularization and constitutive hypoxemia of cancerous tumors. Neoplasia 18, 459-467 (2016)
5.
Roblek, M.* et al.: Targeting of CCL2-CCR2-glycosaminoglycan axis using a CCL2 decoy protein attenuates metastasis through inhibition of tumor cell seeding. Neoplasia 18, 49-59 (2016)
6.
Omar, M. ; Schwarz, M. ; Soliman, D. ; Symvoulidis, P. & Ntziachristos, V.: Pushing the optical Imaging limits of cancer with multi-frequency-band Raster-Scan Optoacoustic Mesoscopy (RSOM). Neoplasia 17, 208-214 (2015)
7.
Dobosz, M. ; Ntziachristos, V. ; Scheuer, W.* & Strobel, S.*: Multispectral fluorescence ultramicroscopy: Three-dimensional visualization and automatic quantification of tumor morphology, drug penetration, and antiangiogenic treatment response. Neoplasia 16, 1-13 (2014)
8.
Laukens, B.* et al.: Smac mimetic bypasses apoptosis resistance in FADD- or caspase-8-deficient cells by priming for tumor necrosis factor α-induced necroptosis. Neoplasia 13, 971-979 (2011)
9.
Schneider, M. et al.: Absence of glutathione peroxidase 4 affects tumor angiogenesis through increased 12/15-lipoxygenase activity. Neoplasia 12, 254-263 (2010)
10.
Baeten, J. ; Haller, J.* ; Shih, H.* & Ntziachristos, V.: In vivo investigation of breast cancer progression by use of an internal control. Neoplasia 11, 220-227 (2009)
11.
Erkan, M.* et al.: Cancer-stellate cell interactions perpetuate the hypoxia-fibrosis cycle in pancreatic ductal adenocarcinoma. Neoplasia 11, 497-508 (2009)
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