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1.
Pukaj, A.* et al.: A genetic association study identifying cis protein quantitative trait loci in patients with MS. Mult. Scler. J. 29, 1097-1098 (2023)
2.
Knauer-Arloth, J. et al.: Identification of functionally relevant genetic variants associated with multiple sclerosis using deep learning. Mult. Scler. J. 25, 906-907 (2019)
3.
Spadaro, M.* et al.: Pathogenic mechanisms of human autoantibodies against myelin oligodendrocyte glycoprotein. Mult. Scler. J. 23, 976-976 (2017)
4.
Joerg, S.* et al.: Cutaneous sodium storage is a major driver of autoimmune responses in neuroinflammation. Mult. Scler. J. 22, 40-40 (2016)
5.
Reinthaler, E.M.* et al.: Mutations in the gene tripeptidyl peptidase II (TPP2) and multiple sclerosis. Mult. Scler. J. 22, 849-849 (2016)
6.
van Nierop, G.P.* ; Mautner, J. ; Mitterreiter, J.G.* ; Hintzen, R.Q.* & Verjans, G.M.*: Intrathecal CD8 T-cells of multiple sclerosis patients recognize lytic Epstein-Barr virus proteins. Mult. Scler. J. 22, 279-291 (2016)
7.
Srivastava, R.* et al.: Differential glycosylation of KIR4.1 in glia cells affects binding of autoantibodies in multiple sclerosis. Mult. Scler. J. 20, 341-342 (2014)
8.
Schulze-Topphoff, U.* et al.: Spontaneous opticospinal encephalomyelitis and meningeal follicles develop in the absence of antibodies. Mult. Scler. J. 19, 387 (2013)
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