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Pandey, R.C.* ; Michel, S.* ; Schieck, M.* ; Binia, A.* ; Liang, L.M.* ; Klopp, N. ; Franke, A.* ; von Berg, A.* ; Bufe, A.* ; Rietschel, E.* ; Heinzmann, A.* ; Laub, O.* ; Simma, B.* ; Frischer, T.* ; Genuneit, J.* ; Illig, T. ; Kabesch, M.*

Polymorphisms in extracellular signal-regulated kinase family influence genetic susceptibility to asthma.

J. Allergy Clin. Immunol. 131, 1245-1247 (2013)
DOI PMC
Open Access Green möglich sobald Postprint bei der ZB eingereicht worden ist.
Asthma susceptibility loci determined by genome-wide association studies explain only a fraction of asthma's hereditability. Pathway analysis has emerged as a next step toward mining hidden genetic factors in complex diseases. A recent study investigating the association between polymorphisms in Toll-like receptor signaling pathways and asthma revealed that 2 members of the mitogen-activated protein kinase (MAPK) pathway (MAP kinase kinase 3 and extracellular signal-regulated kinase 2 [ERK2]) showed strong association with the disease
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Publikationstyp Artikel: Journalartikel
Dokumenttyp Wissenschaftlicher Artikel
ISSN (print) / ISBN 0091-6749
e-ISSN 1097-6825
Zeitschrift The journal of allergy and clinical immunology
Quellenangaben Band: 131, Heft: 4, Seiten: 1245-1247 Artikelnummer: , Supplement: ,
Verlag Elsevier
Verlagsort Amsterdam [u.a.]
Begutachtungsstatus Peer reviewed
Institut(e) Research Unit Molecular Epidemiology (AME)