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21.
Schreiber, S. ; Honz, M. ; Schiemann, M.* ; Protzer, U. & Wisskirchen, K.: Generating CD4+T cells for the T-cell therapy of HBV infection. J. Virol. 25, 47-47 (2018)
22.
Stadler, D. et al.: Identification of ISG20 as the nuclease involved in Interferon-induced decline of HBV cccDNA. J. Virol. 25, 17-17 (2018)
23.
Suffner, S. et al.: Domains of the hepatitis B virus small surface protein S mediating oligomerization. J. Virol. 92:e02232-17 (2018)
24.
Albanese, M. ; Tagawa, T. ; Buschle, A. & Hammerschmidt, W.: MicroRNAs of Epstein-Barr virus control innate and adaptive anti-viral immunity. J. Virol. 91:e01667-16 (2017)
25.
Inturi, R.* ; Mun, K.* ; Singethan, K. ; Schreiner, S. & Punga, T.*: Human adenovirus infection causes cellular E3 ubiquitin ligase MKRN1 degradation involving the viral core protein pVII. J. Virol. 92:e01154-17 (2017)
26.
Shinde, P.V.* et al.: Tumor necrosis factor-mediated survival of CD169+ cells promotes immune activation during vesicular stomatitis virus infection. J. Virol. 92, DOI: 10.1128/JVI.01637-17 (2017)
27.
Bayer, K. ; Banning, C.* ; Bruss, V. ; Wiltzer-Bach, L.* & Schindler, M.: Hepatitis C virus is released via a non-canonical secretory route. J. Virol. 90, 10558-10573 (2016)
28.
Brinkmann, C.* et al.: Tetherin antagonism by the Ebola virus glycoprotein requires an intact receptor-binding domain and can be blocked by GP1-specific antibodies. J. Virol. 90, 11075-11086 (2016)
29.
Chachage, M.* et al.: CD25+ FoxP3+ memory CD4 T cells are frequent targets of HIV infection in vivo. J. Virol. 90, 8954-8967 (2016)
30.
Berrios, C.* et al.: Malawi polyomavirus is a prevalent human virus that interacts with known tumor suppressors. J. Virol. 89, 857-862 (2015)
31.
Bürck, C.* et al.: KAP1 is a host restriction factor that promotes HAdV E1B-55K SUMO modification. J. Virol. 90, 930-946 (2015)
32.
Cui, X.* ; Luckenbaugh, L.* ; Bruss, V. & Hu, J.J.*: Alteration of mature nucleocapsid and enhancement of covalently closed circular DNA formation by Hepatitis B Virus core mutants defective in complete virion formation. J. Virol. 89, 10064-10072 (2015)
33.
Gnirß, K.* et al.: Tetherin sensitivity of influenza A viruses is strain specific: Role of hemagglutinin and neuraminidase. J. Virol. 89, 9178-9188 (2015)
34.
Gondim, M.V. et al.: AP-2 is the crucial clathrin adaptor protein for CD4 downmodulation by HIV-1 Nef in primary infected CD4+ T cells. J. Virol. 89, 12518-12524 (2015)
35.
Hoh, A.* et al.: HBV-infected HepG2hNTCP cells serve as a novel immunological tool to analyze the antiviral efficacy of CD8+ T cells in vitro. J. Virol. 89, 7433-7438 (2015)
36.
Lambelé, M.* et al.: Vpu is the main determinant for tetraspanin downregulation in HIV-1 infected cells. J. Virol. 89, 3247-3255 (2015)
37.
Orabi, A. ; Bieringer, M. ; Geerlof, A. & Bruss, V.: An aptamer against the matrix binding domain on the hepatitis B virus capsid impairs virion formation. J. Virol. 89, 9281-9287 (2015)
38.
Steinbrück, L. et al.: K1 and K15 of Kaposi sarcoma-associated herpes virus are partial functional homologues of latent membrane protein 2A of Epstein-Barr virus. J. Virol. 89, 7248-7261 (2015)
39.
Thiele, F. et al.: MVA-infected dendritic cells present CD4+ T-cell epitopes by endogenous MHC class II presentation pathways. J. Virol. 98, 2698-2709 (2015)
40.
Vassena, L.* et al.: HIV-1 NEF and VPU interfere with L-selectin (CD62L) cell surface expression to inhibit adhesion and signaling in infected CD4+ t lymphocytes. J. Virol. 89, 5687-5700 (2015)
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