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1.
Baumeister, T.* et al.: Anti-inflammatory chemoprevention attenuates the phenotype in a mouse model of esophageal adenocarcinoma. Carcinogenesis 42, 1068-1078 (2021)
2.
Mishra, A.* et al.: Generation of focal mutations and large genomic deletions in the pancreas using inducible in vivo genome editing. Carcinogenesis 41, 334-344 (2020)
3.
Castelletti, N. et al.: Risk of lung adenocarcinoma from smoking and radiation arises in distinct molecular pathways. Carcinogenesis 40, 1240-1250 (2019)
4.
Giopanou, I.* et al.: Osteopontin drives KRAS-mutant lung adenocarcinoma. Carcinogenesis 41, 1134-1144 (2019)
5.
Kanellakis, N.I.* et al.: Tobacco chemical-induced mouse lung adenocarcinoma cell lines pin the prolactin orthologue proliferin as a lung tumour promoter. Carcinogenesis 40, 1352-1362 (2019)
6.
Li, Y.* et al.: Genome-wide interaction study of smoking behavior and non-small cell lung cancer risk in Caucasian population. Carcinogenesis 39, 336-346 (2018)
7.
Liu, H.* et al.: Functional variants in DCAF4 associated with lung cancer risk in European populations. Carcinogenesis 38, 541-551 (2017)
8.
Kaiser, J.C. et al.: Integration of a radiation biomarker into modelling of thyroid carcinogenesis and post-Chernobyl risk assessment. Carcinogenesis 37, 1152-1160 (2016)
9.
Kang, X.* et al.: Polymorphisms of the centrosomal gene (FGFR1OP) and lung cancer risk: A meta-analysis of 14 463 cases and 44 188 controls. Carcinogenesis 37, 280-289 (2016)
10.
Sigurdson, A.J.* et al.: Selected single-nucleotide polymorphisms in FOXE1, SERPINA5, FTO, EVPL, TICAM1 and SCARB1 are associated with papillary and follicular thyroid cancer risk: Replication study in a German population. Carcinogenesis 37, 677-684 (2016)
11.
Walsh, K.M.* et al.: Common genetic variants associated with telomere length confer risk for neuroblastoma and other childhood cancers. Carcinogenesis 37, 576-582 (2016)
12.
Brenner, D.R.* et al.: Identification of lung cancer histology-specific variants applying Bayesian framework variant prioritization approaches within the TRICL and ILCCO consortia. Carcinogenesis 36, 1314-1326 (2015)
13.
Selmansberger, M. et al.: Dose-dependent expression of CLIP2 in post-Chernobyl papillary thyroid carcinomas. Carcinogenesis 36, 748-756 (2015)
14.
Selmansberger, M. et al.: Genomic copy number analysis of Chernobyl papillary thyroid carcinoma in the Ukrainian-American Cohort. Carcinogenesis 36, 1381-1387 (2015)
15.
Kundrát, P. ; Bauer, G.* ; Jacob, P. & Friedland, W.: Mechanistic modelling suggests that the size of preneoplastic lesions is limited by intercellular induction of apoptosis in oncogenically transformed cells. Carcinogenesis 33, 253-259 (2012)
16.
Rosenberger, A.* et al.: Asthma and lung cancer risk: A systematic investigation by the International Lung Cancer Consortium. Carcinogenesis 33, 587-597 (2012)
17.
Frank, B.* et al.: Colorectal cancer and polymorphisms in DNA repair genes WRN, RMI1 and BLM. Carcinogenesis 31, 442-445 (2010)
18.
Frank, B.* et al.: Single nucleotide polymorphisms in Wnt signaling and cell death pathway genes and susceptibility to colorectal cancer. Carcinogenesis 31, 1381-1386 (2010)
19.
Frank, B.* et al.: Association of hydroxyprostaglandin dehydrogenase 15-(NAD) (HPGD) variants and colorectal cancer risk. Carcinogenesis 32, 190-196 (2010)
20.
Hoeft, B.* et al.: Polymorphisms in fatty acid metabolism-related genes are associated with colorectal cancer risk. Carcinogenesis 31, 466-472 (2010)
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