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ß2-adrenoreceptor polymorphisms and asthma.

Lancet 368, 710-711 (2006)
DOI
Open Access Green as soon as Postprint is submitted to ZB.
A database comparison of the aminoacid sequence of the β2-adrenergic receptor from different species predicts in seconds that both the Arg16Gly and Gln27Glu variants are phenotypically benign.1 By contrast, the within-species analysis of the Homo sapiens gene took many years. With the first description in 1993 of all genetic variations of the β2 receptor,2 this receptor immediately became a prime target in many asthma studies.3,4 So why is the current study of Ian Hall and colleagues5 in today's Lancet important?
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Publication type Article: Journal article
Document type Scientific Article
Corresponding Author
ISSN (print) / ISBN 0140-6736
e-ISSN 0099-5355
Journal Lancet, The
Quellenangaben Volume: 368, Issue: 9537, Pages: 710-711 Article Number: , Supplement: ,
Publisher Elsevier
Non-patent literature Publications
Reviewing status Peer reviewed
Institute(s) Institute of Epidemiology (EPI)