PuSH - Publication Server of Helmholtz Zentrum München: Biological monitoring of welders exposed to aluminium.

Information

Clues to quality of journals

Open Access Policy of Helmholtz Association 2016

CC Licencences

Publication Metrics

Navigation

Home

Deutsch

EVA: Electronic Publishing

New EVA Application

Research

Advanced Search

Browse by ...

... HMGU-Authors/Consortia

... Organizational Structure

... Journal

... Publication Type

... Research Data

... Arbeitsgruppen

... Publication Year

Publication overview

Statistics

Statistics (last 5 years)

OA Publications

Publish

Submit Publication

Import publication from...

...EVA

Report missing publication

Highlights

Support & Contact

Contact persons

Help

Data protection

Helmholtz Open Science

JANE Journal Estimator

SHERPA/RoMEO

DOAJ

Export:

Text

Endnote (RIS) BIB

BibTeX

Rossbach, B.* ; Buchta, M.* ; Csanády, G.A. ; Filser, J.G. ; Hilla, W.* ; Windorfer, K.* ; Stork, J.* ; Zschiesche, W.* ; Gefeller, O.* ; Pfahlberg, A.* ; Schaller, K.H.* ; Egerer, E.* ; Pinzón, L.C.* ; Letzel, S.*

Biological monitoring of welders exposed to aluminium.

Toxicol. Lett. 162, 239-245 (2006)

DOI

Open Access Green as soon as Postprint is submitted to ZB.

Abstract
Metrics
Extra information

To evaluate an adequate strategy for biological monitoring of aluminium (Al), a group of 62 Al welders (age in 1999: 23-51 years, median 35 years) was surveyed annually from 1999 to 2003 by determination of pre- and post-shift Al in urine and plasma. Biomonitoring was supplemented by personal air measurements of the total dust concentration. The welders' internal exposure was compared to the exposure of 60 non-exposed assembly workers (age in 1999: 21-51 years, median: 36 years) who were surveyed in 1999, 2001 and 2003. Having a nearly constant dust exposure, median concentrations of Al in urine (Al in plasma) of the welders decreased from 40.1 μg/g to 19.8 μg/g creatinine (8.7 to 4.6 μg/l). For the control group the median levels of Al in urine (plasma) ranged from 4.8 μg/g to 5.2 μg/g creatinine (2.4-4.3 μg/l) indicating a higher sensitivity for the marker Al in urine. No systematic differences have been found between pre- and post-shift internal exposure. This might be caused by the slow elimination kinetics and low systemic bioavailability of Al. A correlation analysis did not yield close relationships between dust exposure, Al in plasma and Al in urine underlining the importance of biomonitoring for assessment of Al exposure. © 2005 Elsevier Ireland Ltd. All rights reserved.

Impact Factor

Scopus SNIP

Web of Science
Times Cited

Scopus
Cited By

Altmetric

2.430

0.000