Clues to quality of journals
Open Access Policy of Helmholtz Association 2016
CC Licencences
Publication Metrics
Home
Deutsch
New EVA Application
Advanced Search
Browse by ...
Publication overview
Statistics (last 5 years)
OA Publications
Submit Publication
Import publication from...
Report missing publication
Contact persons
Help
Text
Endnote (RIS)
BibTeX
Publ. Version/Full Text Volltext
PMC
 |
Open Access Gold |
as soon as is submitted to ZB.
The tumor suppressor p53 connects ribosome biogenesis to cell cycle control: A double-edged sword.
Oncotarget 1, 43-47 (2010)
Since its first description more than 30 years ago p53 has become a paradigm for a protein with versatile functions. P53 sensitizes a large variety of genetic alterations and has been entitled the guardian of the genome. Stabilization of p53 upon DNA damage is accompanied by a complex pattern of modifications, which ascertain the cellular response either in the direction of a reversible or irreversible cell cycle arrest or programmed cell death. More recently it became evident that p53 also responds to non-genotoxic cell stress, in particular if ribosome biogenesis is affected.
Impact Factor
Scopus SNIP
Web of Science
Times Cited
Times Cited
Altmetric
0.000
0.000
20
Annotations
Special Publikation
Hide on homepage
Publication type
Article: Journal article
Document type
Scientific Article
Keywords
PEBOW-COMPLEX; DNA-DAMAGE; PROTEIN; MUTATIONS; DISRUPTION; RPS14; BOP1; PES1; PROLIFERATION; STABILIZATION
Language
english
Publication Year
2010
HGF-reported in Year
0
ISSN (print) / ISBN
1949-2553
e-ISSN
1949-2553
Journal
OncoTarget
Quellenangaben
Volume: 1,
Issue: 1,
Pages: 43-47
Publisher
Impact Journals LLC
Reviewing status
Peer reviewed
POF-Topic(s)
30504 - Mechanisms of Genetic and Environmental Influences on Health and Disease
Research field(s)
Immune Response and Infection
PSP Element(s)
G-501490-001
PubMed ID
21293052
WOS ID
000293475100007
Erfassungsdatum
2010-11-16