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Schumann, T. ; König, J.* ; von Loeffelholz, C.* ; Vatner, D.F.* ; Zhang, D.* ; Perry, R.J.* ; Bernier, M.* ; Chami, J.* ; Henke, C. ; Kurzbach, A. ; El-Agroudy, N.N. ; Willmes, D.M. ; Pesta, D.* ; de Cabo, R.* ; O Sullivan, J.F.* ; Simon, E.* ; Shulman, G.I.* ; Hamilton, B.S.* ; Birkenfeld, A.L.

Publisher Correction: Deletion of the diabetes candidate gene Slc16a13 in mice attenuates diet-induced ectopic lipid accumulation and insulin resistance.

Comm. Biol. 4:890 (2021)
Correction DOI PMC
Creative Commons Lizenzvertrag
Open Access Gold as soon as Publ. Version/Full Text is submitted to ZB.
The original published version of the Article contained an error in the abstract whereby the words “loss of” were inadvertently omitted from the following sentence: “We show that loss of Slc16a13 increases mitochondrial respiration in the liver, leading to reduced hepatic lipid accumulation and increased hepatic insulin sensitivity in high-fat diet fed Slc16a13 knockout mice.” The error has been corrected in the HTML and PDF versions of the Article.
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Publication type Other: Correction, Addition
Corresponding Author
ISSN (print) / ISBN 2399-3642
e-ISSN 2399-3642
Journal Communications Biology
Quellenangaben Volume: 4, Issue: 1, Pages: , Article Number: 890 Supplement: ,
Publisher Springer
Publishing Place London
Non-patent literature Publications
Reviewing status Peer reviewed
Institute(s) Institute of Diabetes Research and Metabolic Diseases (IDM)
Institute for Pancreatic Beta Cell Research (IPI)
Helmholtz AI - DLR (HAI - DLR)