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Schumann, T. ; König, J.* ; von Loeffelholz, C.* ; Vatner, D.F.* ; Zhang, D.* ; Perry, R.J.* ; Bernier, M.* ; Chami, J.* ; Henke, C. ; Kurzbach, A. ; El-Agroudy, N.N. ; Willmes, D.M. ; Pesta, D.* ; de Cabo, R.* ; O Sullivan, J.F.* ; Simon, E.* ; Shulman, G.I.* ; Hamilton, B.S.* ; Birkenfeld, A.L.

Publisher Correction: Deletion of the diabetes candidate gene Slc16a13 in mice attenuates diet-induced ectopic lipid accumulation and insulin resistance.

Comm. Biol. 4:890 (2021)
Correction DOI PMC
Open Access Gold
Creative Commons Lizenzvertrag
The original published version of the Article contained an error in the abstract whereby the words “loss of” were inadvertently omitted from the following sentence: “We show that loss of Slc16a13 increases mitochondrial respiration in the liver, leading to reduced hepatic lipid accumulation and increased hepatic insulin sensitivity in high-fat diet fed Slc16a13 knockout mice.” The error has been corrected in the HTML and PDF versions of the Article.
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Publication type Other: Correction, Addition
Language english
Publication Year 2021
HGF-reported in Year 2021
ISSN (print) / ISBN 2399-3642
e-ISSN 2399-3642
Quellenangaben Volume: 4, Issue: 1, Pages: , Article Number: 890 Supplement: ,
Publisher Springer
Publishing Place London
Reviewing status Peer reviewed
Institute(s) Institute of Diabetes Research and Metabolic Diseases (IDM)
Institute of Pancreatic Islet Research (IPI)
Helmholtz AI - DLR (HAI - DLR)
POF-Topic(s) 90000 - German Center for Diabetes Research
Research field(s) Helmholtz Diabetes Center
PSP Element(s) G-502400-001
G-502600-012
Scopus ID 85109859139
PubMed ID 34262128
Erfassungsdatum 2021-07-22