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P(URI)fying novel drivers of NASH and HCC: A feedforward loop of IL17A via white adipose tissue.
Cancer Cell 30, 15-17 (2016)
How obesity and metabolic syndrome trigger non-alcoholic steatohepatitis (NASH) and hepatocellular carcinoma (HCC) remains elusive. In this issue, Gomes and colleagues describe that nutrient surplus induces hepatic URI expression, triggering genotoxicity and IL17A expression, thus leading to insulin resistance, NASH, and HCC. IL17A signaling blockers might become a readily translatable therapy.
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Publication type
Article: Journal article
Document type
Editorial
Keywords
Hepatocellular-carcinoma; Liver; Tumorigenesis; Epidemiology; Prevention; Cancer; Cells; Uri
Language
english
Publication Year
2016
HGF-reported in Year
2016
ISSN (print) / ISBN
1535-6108
e-ISSN
1878-3686
Journal
Cancer Cell
Quellenangaben
Volume: 30,
Issue: 1,
Pages: 15-17
Publisher
Cell Press
Publishing Place
Cambridge, Mass.
Reviewing status
Peer reviewed
Institute(s)
Institute of Virology (VIRO)
POF-Topic(s)
30504 - Mechanisms of Genetic and Environmental Influences on Health and Disease
Research field(s)
Immune Response and Infection
PSP Element(s)
G-551600-001
WOS ID
WOS:000380077100008
PubMed ID
27411585
Erfassungsdatum
2016-07-26